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Hexahydrocannabiphorol ( HHCP, sometimes mistakenly referred to as hexahydroxycannabiphorol) is a semi-synthetic cannabinoid derivative which has been marketed since around 2021. [1] [2] It is believed to be made from the hydrogenation of tetrahydrocannabiphorol (THCP). THCP is only reported as a trace component of cannabis in 2019. [3]
Not a lot of data exists about how HHC affects your body. But it is a form of THC, and those short-term effects are well-known. THC can: Alter your senses, including your sense of time. Change ...
Drop shipping is a form of retail business in which the seller accepts customer orders without keeping stock on hand. Instead, in a form of supply chain management, the seller transfers the orders and their shipment details either to the manufacturer, a wholesaler, another retailer, or a fulfillment house, which then ships the goods directly to ...
dry mouth. increased heart rate. low blood pressure. memory loss. red eyes. slower reaction times. tiredness. Because of the lack of research on HHC, we don’t know whether it can produce some or ...
Find out what you need to know about Candida auris, a fungal infection affecting people in hospitals. Mole crickets are common pests that wreak havoc on lawns and turf. Learn the signs of a mole ...
HHCP-O-acetate ( HHCPO, HHCP-O) is a semi-synthetic derivative of tetrahydrocannabiphorol (THCP) derived in several steps by hydrogenation to hexahydrocannabiphorol (HHCP) followed by acetylation of the OH group. It has been found as a component of grey-market cannabis products such as e-cigarette liquids and edible gumdrops, and is allegedly a ...
Hexahydrocannabinol ( HHC) is a hydrogenated derivative of tetrahydrocannabinol (THC). It is a naturally occurring phytocannabinoid that has rarely been identified as a trace component in Cannabis sativa, [1] [2] but can also be produced synthetically by acid cyclization and hydrogenation of tetrahydrocannabinol.
Hereditary haemochromatosis type 1 ( HFE-related Hemochromatosis) [3] is a genetic disorder characterized by excessive intestinal absorption of dietary iron, resulting in a pathological increase in total body iron stores. [4] Humans, like most animals, have no mechanism to regulate excess iron, simply losing a limited amount through various ...
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